Job Description
The project is based on the hypothesis that activation of the complement system—a critical component of the innate immune response—plays a pivotal role in the initiation and progression of PH. This systemic activation results in local inflammation and vascular remodeling, characteristics that are central to the pathogenesis of PH. The research aims to dissect the molecular mechanisms by which the complement system influences these pathological changes and to explore targeted therapeutic strategies that could mitigate these effects. The work utilizes many mouse, rat, and large animal models to investigate various etiologies of PH.
Collaborative Environment:
The postdoc will work within a multidisciplinary team comprising experts in pulmonary vascular biology, complement biology, immunology, and proteomics. This collaborative setting is designed to foster innovative approaches and rapid translation of research findings from bench to bedside.